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And then from there [ Mukerji actively avoids typecasting, and has been credited in the media for her versatility.

At the peak of her career, Mukerji featured in listings of the most attractive Indian celebrities, was one of the highest-paid actresses in Bollywood, and the brand ambassador for a number of products.

From Wikipedia, the free encyclopedia. Indian film actress. Bombay , Maharashtra , India. Aditya Chopra. Ram Mukherjee father. See also: Mukherjee-Samarth family.

See also: Rani Mukerji filmography. Main article: List of awards and nominations received by Rani Mukerji. Bollywood portal Film portal India portal.

Archived from the original on 17 February Retrieved 26 March Archived from the original on 27 December Retrieved 24 April Archived from the original on 21 September Retrieved 5 January Archived from the original on 19 September Retrieved 3 July Retrieved 23 December Retrieved 12 January Archived from the original on 3 January Retrieved 3 January Mid Day.

Archived from the original on 3 April Archived from the original on 19 February The Tribune. Archived from the original on 21 November Retrieved 16 July The Times of India.

Archived from the original on 21 May Retrieved 21 May Archived from the original on 30 October Retrieved 13 January Archived from the original on 26 July Retrieved 6 January Archived from the original on 4 January British Film Institute.

Archived from the original on 2 April Retrieved 19 March Hindustan Times. Archived from the original on 28 June Retrieved 8 March Retrieved 5 May Box Office India.

Archived from the original on 10 October Retrieved 8 January Retrieved 11 February Archived from the original on 30 November Retrieved 27 November Duke University Press.

Archived from the original on 5 June Retrieved 6 May India Today. Archived from the original on 8 August Retrieved 3 November Archived from the original on 18 May Archived from the original on 5 August Archived from the original on 17 October Archived from the original on 15 October Retrieved 9 January Archived from the original on 2 August Retrieved 22 July India Abroad.

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The Hindu. Archived from the original on 7 April Archived from the original on 24 October Bollywood Hungama.

Archived from the original on 31 December Retrieved 23 July Archived from the original on 16 August The Guardian. Archived from the original on 10 May Retrieved 25 April Archived from the original on 24 January Retrieved 25 January Archived from the original on 14 October Retrieved 11 November Archived from the original on 25 January Archived from the original on 6 July Archived from the original on 13 November So am I".

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Archived from the original on 10 December Retrieved 22 September Rotten Tomatoes. Retrieved 25 December Gupta, Pratim 24 June Retrieved 6 October Thomson Living Media India Limited.

Archived from the original on 27 June Retrieved 14 July Archived from the original on 26 April Archived from the original on 29 May The Economic Times.

Retrieved 22 May Archived from the original on 5 April Retrieved 17 October Daily News and Analysis. Archived from the original on 11 February Retrieved 25 October Retrieved 17 December Archived from the original on 9 January Retrieved 22 April Retrieved 31 January Retrieved 12 October Archived from the original on 2 June Retrieved 21 January Archived from the original on 15 March Archived from the original on 19 January Archived from the original on 1 December Retrieved 28 January The National.

Archived from the original on 5 May Archived from the original on 7 May Retrieved 23 August Archived from the original on 1 August Retrieved 1 August Deccan Chronicle.

Archived from the original on 30 July Archived from the original on 25 August Retrieved 23 April Archived from the original on 20 January Retrieved 20 January Archived from the original on 2 March You may need urgent medical attention.

Before starting any new medicine, tell your doctor or pharmacist that you are taking Rani 2. If you plan to have surgery, including dental surgery, tell your doctor or dentist that you are taking Rani 2.

Be careful driving or operating machinery until you know how Rani 2 affects you. Rani 2 generally does not cause any problems with your ability to drive a car or operate machinery.

However, as with many other medicines, Rani 2 may cause dizziness or light-headedness in some people. Some self-help measures suggested below may help your condition.

Talk to your doctor or pharmacist about these measures and for more information. Tell your doctor or pharmacist as soon as possible if you do not feel well while you are taking Rani 2.

Like all other medicines, Rani 2 may have unwanted side effects in some people. Sometimes they are serious, most of the time they are not.

You may need medical treatment if you get some of the side effects. Do not be alarmed by this list of possible side effects. You may not experience any of them.

Tell your doctor immediately or go to Accident and Emergency at the nearest hospital if you notice any of the following:.

Some health problems may arise from the condition being treated itself, rather than the treatment. For this reason, contact your doctor immediately if you notice any of the following:.

Keep Rani 2 where children cannot reach it. A locked cupboard at least one-and-a-half metres above the ground is a good place to store medicines.

Keep your tablets in the pack until it is time to take them. If you take the tablets out of the pack, they will not keep well. Do not store Rani 2 or any other medicine in the bathroom or near a sink.

Do not leave Rani 2 in the car or on windowsills. Heat and dampness can destroy some medicines. If your doctor tells you to stop taking Rani 2, or your tablets have passed its expiry date, ask your pharmacist what to do with any that are left over.

Ranitidine hydrochloride is a white or pale yellow, crystalline powder, freely soluble in water and in methanol, sparingly soluble in ethanol, very slightly soluble in methylene chloride.

Ranitidine is an aminoalkyl-substituted furan and is structurally different from cimetidine lacking the imidazole ring and the cyanoguanidine group.

Chemical structure. Chemical name: N- 2- 5- dimethylamino methyl furan-yl methyl thio ethyl -N'- methyl nitro-1,1-ethenediamine hydrochloride.

Molecular weight: CAS number. Each Rani 2 tablet contains ranitidine hydrochloride as the active ingredient equivalent to mg or mg of ranitidine.

For the full list of excipients, see Section 6. Rani 2 mg. Creamish yellow, biconvex, round, film-coated tablets with "G" on one side and "00" over "30" on the reverse.

Creamish yellow, biconvex, capsule shaped, film-coated tablets with "G" on one side and "" on the reverse. Mechanism of action.

Ranitidine hydrochloride is a histamine H 2 -receptor antagonist. Animal experiments both in vitro and in vivo have established that ranitidine is a selective, competitive antagonist of histamine at H 2 -receptor sites.

Ranitidine has no significant interaction at histamine H 1 -receptors, muscarinic receptors or beta adrenoceptors. Ranitidine is a potent inhibitor of gastric secretion in the rat and dog.

Clinical trials. All the evidence from human studies is compatible with a selective, competitive antagonism of histamine H 2 -receptors by ranitidine in man.

Oral or intravenous administration of ranitidine inhibits both basal gastric secretions and gastric acid secretion induced by histamine, pentagastrin and other secretagogues.

On a weight basis ranitidine is between four and nine times more potent than cimetidine. After oral administration of ranitidine, the plasma concentrations of ranitidine achieved are directly related to the dose administered.

Pepsin secretion is also inhibited by ranitidine, but secretion of gastric mucus is not affected. Ranitidine does not alter the secretion of bicarbonate or enzymes from the pancreas in response to secretin and pancreozymin.

Reduction in gastric acid secretion induced by ranitidine mg twice daily for seven days did not cause bacterial overgrowth in the stomach.

Pulse rate, blood pressure, electrocardiogram and electroencephalogram were not significantly affected in man following recommended doses of ranitidine.

One study in 30 male duodenal ulcer patients showed a significant decrease in basal thyroxine levels after 4 weeks treatment with mg ranitidine daily, but no significant change in thyroid stimulating hormone was noted.

Acute administration of 50 mg ranitidine intravenously had no effect on plasma aldosterone in healthy male volunteers whereas it caused a significant reduction in vasopressin.

Cimetidine mg intravenously had a similar effect on vasopressin. Peak plasma levels occur about two to three hours after oral administration of ranitidine.

Absorption is not significantly altered by food or concurrent antacid administration. Two distinct peaks or a plateau in the absorption phase suggest reabsorption of drug secreted into the intestine.

The elimination half-life is approximately two hours. Plasma concentrations decline bi-exponentially, with a terminal half-life of 2 to 3 hours.

The major route of elimination of unchanged ranitidine is renal. Patients over 50 years of age. In patients over 50 years of age, half-life is prolonged 3 to 4 hours and clearance is reduced, consistent with the age-related decline of renal function.

This difference exceeds the effect of declining renal function, and indicates increased bioavailability in older patients.

Impairment of renal function requires a reduction in dosage see Section 4. Impairment of liver function may increase the bioavailability of ranitidine but has no significant effect on the elimination half-life.

However, in the presence of normal renal function, no dosage reduction for oral ranitidine appears necessary in patients with hepatic impairment.

Short-term treatment of proven duodenal ulcer and gastric ulcer. Maintenance treatment to reduce the risk of relapse in duodenal ulcer.

Maintenance treatment for periods up to one year to reduce the risk of relapse in patients with documented healing of benign gastric ulcer.

Treatment of gastrinoma Zollinger-Ellison syndrome. Short-term symptomatic treatment of reflux oesophagitis unresponsive to conservative antireflux measures and simple drug therapies such as antacids.

Maintenance treatment to reduce the risk of relapse of reflux oesophagitis. Treatment of scleroderma oesophagitis. Patients with known hypersensitivity to ranitidine hydrochloride or any components of Rani 2 tablets.

Gastric ulcer. Treatment with a histamine H 2 -antagonist may mask symptoms associated with carcinoma of the stomach and, therefore, may delay diagnosis of the condition.

Accordingly, where gastric ulcer is suspected, the possibility of malignancy should be excluded before therapy with ranitidine is instituted.

Long-term use. The risk of ulcer recurrence is determined by many factors. Evidence from controlled clinical trials of up to 18 months continuous treatment with ranitidine has not revealed any undue untoward effects.

Rare clinical reports suggest that ranitidine may precipitate acute porphyric attacks. Ranitidine should therefore be avoided in patients with a history of acute porphyria.

Gastric pH. Agents that elevate gastric pH may increase the already present risk of nosocomial pneumonia in intubated intensive care patients receiving mechanical ventilation.

In patients such as the elderly, persons with chronic lung disease, diabetes or the immunocompromised, there may be an increased risk of developing community acquired pneumonia.

A large epidemiological study showed an increased risk of developing community acquired pneumonia in current users of H 2 -receptor antagonists versus those that had stopped treatment, with an observed adjusted relative risk of 1.

Use in renal impairment. Ranitidine is excreted via the kidneys and in the presence of renal impairment plasma levels of ranitidine are increased and prolonged.

Accordingly in the presence of significant renal impairment, serum levels should be monitored and dosage adjustments made.

The clearance of ranitidine is increased during haemodialysis. Use in the elderly. No data available. Paediatric use. Experience with ranitidine tablets in children is limited and such use has not been fully evaluated in clinical studies.

It has however, been used successfully in children aged 8 to 18 years in doses up to mg twice daily. Effects on laboratory tests.

Ranitidine has the potential to affect the absorption, metabolism or renal excretion of other drugs. The altered pharmacokinetics may necessitate dosage adjustment of the affected drug or discontinuation of treatment.

Interactions occur by several mechanisms including: 1. Inhibition of cytochrome Plinked mixed function oxygenase system.

Ranitidine at usual therapeutic doses does not potentiate the actions of drugs which are inactivated by this enzyme system such as diazepam, lidocaine, phenytoin, propranolol and theophylline.

There have been reports of altered prothrombin time with coumarin anticoagulants e. Due to the narrow therapeutic index, close monitoring of increased or decreased prothrombin time is recommended during concurrent treatment with ranitidine.

Competition for renal tubular secretion. Since ranitidine is partially eliminated by the cationic system, it may affect the clearance of other drugs eliminated by this route.

High doses of ranitidine e. Alteration of gastric pH. The bioavailability of certain drugs may be affected. This can result in either an increase in absorption e.

If high doses 2 g of sucralfate are coadministered with ranitidine the absorption of the latter may be reduced. This effect is not seen if sucralfate is taken after an interval of two hours.

Effects on fertility. There are no data on the effects of ranitidine on human fertility. Reproduction studies performed in rats and rabbits have revealed no evidence of impaired fertility or harm to the foetus due to ranitidine.

Category B1 The safety of ranitidine in pregnancy has not been established. Ranitidine crosses the placenta and should only be used during pregnancy if considered essential.

If the administration of ranitidine is considered to be necessary, its use requires that the potential benefits be weighed against possible hazards to the patient and to the foetus.

Ranitidine is secreted in breast milk in lactating mothers, but the clinical significance of this has not been fully evaluated.

Ranitidine should only be used by breastfeeding mothers if considered essential. The following have been reported as events in clinical trials or in the routine management of patients treated with ranitidine.

The relationship to ranitidine therapy has not been clear in many cases. Headache, sometimes severe, has been reported in a very small proportion of patients.

Central nervous system. Rarely, malaise, dizziness, somnolence, insomnia and vertigo. Rare cases of reversible mental confusion, depression and hallucinations have been reported, predominantly in severely ill and elderly patients.

In addition, reversible involuntary movement disorders have been reported rarely. There have been a few reports of reversible blurred vision suggestive of a change in accommodation.

Reversible impotence has been reported rarely. As with other H 2 -receptor antagonists rare reports of tachycardia, bradycardia, premature ventricular beats, atrioventricular block, and asystole.

Rare reports of arthralgias and myalgia. Rare reports of agranulocytosis or pancytopenia, sometimes with marrow hypoplasia or aplasia have been reported.

Blood count changes leucopenia, thrombocytopenia have occurred in a few patients. These are usually reversible.

Controlled studies in animals and man have shown no stimulation of any pituitary hormone by ranitidine, no antiandrogenic activity, and cimetidine induced gynaecomastia and impotence in hypersecretory patients have resolved when ranitidine was substituted.

However, occasional cases of breast conditions such as gynaecomastia and galactorrhoea, impotence and loss of libido have been reported in male patients receiving ranitidine but the incidence did not differ from that in the general population.

Rash including rare cases of mild erythema multiforme. Rare cases of vasculitis and alopecia have been reported.

Transient and reversible changes in the liver function tests can occur. In normal volunteers, SGPT values were increased to at least twice the pretreatment levels in 6 of 12 subjects receiving mg intravenously four times daily for 7 days, and in 4 of 24 subjects receiving 50 mg intravenously four times daily for 5 days.

There have been occasional reports of hepatitis, hepatocellular or hepatocanalicular or mixed, with or without jaundice. These were usually reversible.

Very rare cases of acute interstitial nephritis have been reported. Rare cases of hypersensitivity reactions e.

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